Northwestern University Feinberg School of Medicine
Feinberg Cardiovascular Research Institute

News, Announcements, and Awards

Read the latest news from the Feinberg Cardiovascular Research Institute. The links below take you to articles where you can learn more about our latest achievements and news, as well as our investigators' awards and honors.

  • 11.2.2017
    David Barefield, PhD, Elizabeth McNally, MD, PhD, and Lisa Wilsbacher, MD, PhD are all authors on a new study published in Circulation which describes a mutation in a newly discovered gene that increases the risk of arrhythmia and cardiomyopathy.  Read the story from Northwestern's News Center here.
  • 11.02.2017
    In three recent publications, Northwestern Medicine students and trainees demonstrated the potential of targeting inflammatory pathways in order to limit tissue damage and improve repair after a heart attack. The research was led by Edward Thorp, PhD, associate professor of Pathology and a member of the Feinberg Cardiovascular Research Institute (FCVRI).  Read the article from the Northwestern News Center here.
  • 10.18.2017
    Hossein Ardehali, MD, PhD, professor of Medicine in the Division of Cardiology, has been named the new director of Feinberg’s Medical Scientist Training Program (MSTP), effective September 1.   Xunrong Luo, MD, PhD, the Margaret Gray Morton Professor of Medicine in the Division of Nephrology/Hypertension, was named associate director of MSTP Admissions.
  • 08.17.2017

    A team of scientists has discovered that mutations in the genes FOXC2 and GJC2 are associated with defects in venous valves, flaps within veins that help maintain proper blood flow.

    The study, published in the Journal of Experimental Medicineand led by investigators at King’s College London, was co-authored by Tsutomu Kume, PhD, professor of Medicine in the Division of Cardiology and of Pharmacology.

  • 08.11.2017

    A team of scientists has discovered that mutations in the genes FOXC2 and GJC2 are associated with defects in venous valves, flaps within veins that help maintain proper blood flow.

    The study, published in the Journal of Experimental Medicineand led by investigators at King’s College London, was co-authored by Tsutomu Kume, PhD, professor of Medicine in the Division of Cardiology and of Pharmacology.

  • 05.17.2017
    A new study, published in Science Signaling, provides a surprising preventive role of reactive oxygen species (ROS) against preeclampsia. Preeclampsia is a pregnancy complication characterized by the acute onset of hypertension and proteinuria. Preeclampsia accounts for 10-15% of maternal death and around 4-5% stillbirth all over the world. A Northwestern scientist, Dr. Tomokazu Souma (Quaggin Lab) in collaboration with researchers in Tohoku University, University of Tsukuba, and Harvard medical school identified beneficial/preventive role of reactive oxygen species (ROS) in pathogenesis of preeclampsia using a mouse model. Increasing oxidative stress by depleting a cellular antioxidant stress pathway improves both maternal and fetal outcomes. Conversely, reducing oxidative stress worsens the outcomes. The results may explain the largely negative results of clinical trials for antioxidants in prevention of preeclampsia. Read the paper in Science Signaling